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Ferroptosis has been implicated as an important process not only in health but also in diseases, including cancer, COVID-19, hematologic, neurodegenerative, cardiac, kidney, infectious diseases, and many others [71,72,73,74,75,76,77,78,79,80]
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Accordingly, this review synthesizes contemporary evidence on the major drivers of ethanol-induced neurotoxicity, including oxidative injury, mitochondrial dysfunction, protein misfolding, neuroinflammation, calcium dysregulation, apoptosis, and epigenetic alterations
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