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GPX4-independent ferroptosis—a new strategy in disease's therapy | Cell Death Discovery
U937 cells infected with wild-type or UV-inactivated HSV-1 displayed inhibition of Fas-induced apoptosis, suggesting that a structural viral component may be exerting this effect (Marino-Merlo et al., 2016)
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shows that a combinatorial complex between estrogen receptor (ER)-β and endothelial nitric oxide synthase (eNOS) could repress transcription of prognostic genes that are down-regulated in prostate tumors, such as the glutathione transferase gene GSTP1 (Re et al., 2011)
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