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autism glutathione genes Mercury-induced excitotoxicity in spectrum disorder: disruption of glutamatergic homeostasis and the therapeutic role of the selenium– axis | BioMetals updatedat_2026-02-13T00:00:33
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autism glutathione genes Mercury-induced excitotoxicity in spectrum disorder: disruption of glutamatergic homeostasis and the therapeutic role of the selenium– axis | BioMetals updatedat_2026-02-13T00:00:33
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autism glutathione genes Mercury-induced excitotoxicity in spectrum disorder: disruption of glutamatergic homeostasis and the therapeutic role of the selenium– axis | BioMetals updatedat_2026-02-13T00:00:33
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autism glutathione genes Mercury-induced excitotoxicity in spectrum disorder: disruption of glutamatergic homeostasis and the therapeutic role of the selenium– axis | BioMetals updatedat_2026-02-13T00:00:33
NAC N-Acétylcystéine + Glutathion Fort
A recent study shows that inactivation of PTP1B may also be involved in the activation of JNK/c-Jun pathway [57]
updatedat_2026-02-13T00:00:33
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In the body, it is transformed into both bioactive coenzyme forms: methylcobalamin and adenosylcobalamin
autism glutathione genes Mercury-induced excitotoxicity in spectrum disorder: disruption of glutamatergic homeostasis and the therapeutic role of the selenium– axis | BioMetals updatedat_2026-02-13T00:00:33
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